What Are we Drinking?

Are you trying to kill me? You do know that's poison, right? Who have you been talking to? This is how our uniformed public thinks. If I asked you if you would drink industrial waste, would you?

Chances are your doing as you read this. Its in soft drinks with aspartame. Its in public water with chlorine at least chlorine is proven to clean. Not your teeth but it will handle a pool.

The most damaging environmental pollutant of the cold war was added to our drinking supply by the advice of who? Its been proven to have many harmful effects look at the study of factory workers below. It was first known as a pesticide then as a nerve agent by Nazi Germany. Once it had the intrest of the CIA it was deemed safe after extremely doctored test.

Click this link to find out what you been Drinking in your neighborhood?



1947. Bull Johns Hopkins Hosp. 81:257Ð266.

Grob D, Harvey AM, Langworthy OR, Lilienthal JL Jr.

The administration of di-isopropyl fluorophosphate (DFP) to man, III: Effect on the central nervous system with special reference to the electrical activity of the brain.

As cited in: 1997. Medical Aspects of Chemical and Biological Warfare. Chapter 5 NERVE AGENTS by Frederick R. Sidell, M.D. Published by the Office of The Surgeon General at TMM Publications, Borden Institute, Walter Reed Army Medical Center, Washington, DC 20307-5001.
The fluoresces in aluminum and cryolite workers

The author presents extensive clinical studies on 113 cryolite workers from Polevo and Juzno-Uralsk and from the Eclectrolytic Facility of the aluminum factories in Uralsk Bogoslovskij, Volgograd, Sumgait and Novo-Kuzneck. The cases were divided into three clinical groups which the author believes present the three major stages of fluoresces:

1. The earliest manifestations of fluoresces involve the skin, the upper air passages and the gums. At this stage of the disease, vesicular and ulcerative dermatitis, strophic rhinitis, pharyngitis, laryngitis and periodontal disease appear. Disorders of the stomach and duodenum are not infrequent, associated with pronounced motor and secretory changes which consist at first of a hyper acid and later of an achylic gastritis. In addition, the author observed a fluorogenic decreased activity of several important liver enzymes, especially enolase, cholesterase and serum GOT. Kidney damage is another early symptom as indicated by microlbuminuria and microhematuria. Changes in the nervous system occur, according to the author, by vaso-vegetative dystonia with pronounced hypotonia and bradycardia. Polyneuropathies and radiculitis are common manifestations suggestive of involvement of the peripheral nervous system. The first stage of the disease usually occurs after 4 to 7 years' exposure.

2. With progressive exposure, the disease turns into its second stage which is characterized by changes in bones and tooth enamel, by toxic nephropathias, hepatitis and gastritis. According to the author both osteoporosis and osteosclerosis occur early in the disease. The osteoporosis can become so extensive it may give rise to cystic bone cavities. The long bones of the forearms and of the legs exhibit mainly osteosclerosis. Characteristically, these changes show symmetrical localization. Bony apposition at the endosteum decreases the width of the medullary portion of these bones.

3. The third stage of fluoresces is characterized by generalized osteosclerosis, joint deformities and, at the vertebral column, by ossification of ligaments and tendons. The author considers osteosclerosis the least reversible sign of fluoride intoxication. Even after an interruption of exposure to fluoride of 10 to 12 yeas, she observed further progression of the toxic processes...
In concluding, the author expresses her views concerning the pathogenesis of fluoresces. She considers a functional involvement of the anterior lobe of the hypophysis, combined with a marked insufficiency of the adrenal cortex, the principal mechanism of the disease. As proof for the functional impairment of the hypophysis, she cites her observation that fluorotic patients are intolerant to corticosteroids...


Japan J Public Health 1967; 14:41-47
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